[Inverse correlation between Snail and E-cadherin expression in carcinoma cell lines and invasive ability in vitro]

Zhonghua Zhong Liu Za Zhi. 2006 Jan;28(1):17-20.
[Article in Chinese]

Abstract

Objective: Malignant transformation of epithelial cell frequently coincides with loss of E-cadherin. Here we study the expression of Snail and E-cadherin and correlate their expression with cell differentiation and in vitro invasion.

Methods: The expression and localization of Snail and E-cadherin were studied by Northern blot and laser confocal microscopy in two normal cell lines (MDCK, NIH 3T3) and six carcinoma cell lines (A431, MCF-7, MDA-MB-453, HepG2, MDA-MB-435s, MDA-MB-231). Boyden chamber assay was done to detect the invasive ability of cells in vitro.

Results: Snail mRNA and protein were detected in fibroblasts NIH 3T3 and poorly differentiated carcinoma cell lines HepG2, MDA-MB-435s and MDA-MB-231. On the contrary, E-cadherin mRNA and protein were detected in normal epithelial cell line MDCK and well differentiated carcinoma cell lines A431 and MDA-MB-453. In MCF-7 cells, Snail and E-cadherin expressions were revealed both at mRNA and protein levels. The cells with higher expression of Snail had stronger ability of invasion than those with lower expression of Snail.

Conclusion: There is an inverse correlation between Snail and E-cadherin expressions and their expressions are correlated with cell differentiation and tumor invasiveness.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3T3 Cells / metabolism
  • Animals
  • Cadherins / biosynthesis*
  • Cadherins / genetics
  • Cell Differentiation
  • Cell Line
  • Cell Line, Tumor
  • Cell Transformation, Neoplastic / metabolism*
  • Dogs
  • Epithelial Cells / cytology
  • Epithelial Cells / metabolism*
  • Humans
  • Mice
  • Neoplasm Invasiveness
  • Snail Family Transcription Factors
  • Transcription Factors / biosynthesis*
  • Transcription Factors / genetics

Substances

  • Cadherins
  • Snail Family Transcription Factors
  • Transcription Factors