Effect of insulin on Ca(2+)-dependent hyperpolarization in erythrocytes from healthy donors and patients with type 2 diabetes mellitus accompanied by arterial hypertension

Bull Exp Biol Med. 2005 Nov;140(5):499-501. doi: 10.1007/s10517-006-0007-5.

Abstract

Insulin decreased A23187-induced hyperpolarization of the erythrocyte membrane in healthy donors. These data indicate that insulin plays a role in the regulation of Ca(2+)-activated potassium channels in human erythrocytes. However, insulin had little effect on hyperpolarization response of cells induced by artificial ascorbate--phenazine methosulfate donor-acceptor system. Addition of insulin to cell suspension from patients with type 2 diabetes mellitus did not modulate hyperpolarization of the erythrocyte membrane induced by A23187 or ascorbate-phenazine methosulfate, which reflects impairment of regulatory mechanisms for Ca(2+)-activated potassium channels in erythrocytes.

MeSH terms

  • Arteries / pathology*
  • Ascorbic Acid / chemistry
  • Calcimycin / pharmacology
  • Calcium / metabolism*
  • Carbonyl Cyanide m-Chlorophenyl Hydrazone / pharmacology
  • Case-Control Studies
  • Diabetes Mellitus, Type 2 / metabolism*
  • Erythrocytes / metabolism*
  • Female
  • Humans
  • Hydrogen-Ion Concentration
  • Hypertension / pathology*
  • Insulin / metabolism
  • Insulin / pharmacology*
  • Ionophores / pharmacology
  • Male
  • Methylphenazonium Methosulfate / pharmacology
  • Oxidation-Reduction

Substances

  • Insulin
  • Ionophores
  • Methylphenazonium Methosulfate
  • Calcimycin
  • Carbonyl Cyanide m-Chlorophenyl Hydrazone
  • Ascorbic Acid
  • Calcium