Escherichia coli cytotoxic necrotizing factor 1 blocks cell cycle G2/M transition in uroepithelial cells

Infect Immun. 2006 Jul;74(7):3765-72. doi: 10.1128/IAI.01413-05.

Abstract

Evidence is accumulating that a growing number of bacterial toxins act by modulating the eukaryotic cell cycle machinery. In this context, we provide evidence that a protein toxin named cytotoxic necrotizing factor 1 (CNF1) from uropathogenic Escherichia coli is able to block cell cycle G(2)/M transition in the uroepithelial cell line T24. CNF1 permanently activates the small GTP-binding proteins of the Rho family that, beside controlling the actin cytoskeleton organization, also play a pivotal role in a large number of other cellular processes, including cell cycle regulation. The results reported here show that CNF1 is able to induce the accumulation of cells in the G(2)/M phase by sequestering cyclin B1 in the cytoplasm and down-regulating its expression. The possible role played by the Rho GTPases in the toxin-induced cell cycle deregulation has been investigated and discussed. The activity of CNF1 on cell cycle progression can offer a novel view of E. coli pathogenicity.

MeSH terms

  • Bacterial Toxins
  • Cell Division / physiology*
  • Cell Line, Tumor
  • Cyclin B / metabolism
  • Cyclin B1
  • Cytotoxins / physiology*
  • Escherichia coli / pathogenicity
  • Escherichia coli / physiology*
  • Escherichia coli Proteins / physiology*
  • G2 Phase / physiology*
  • Growth Inhibitors / physiology*
  • Humans
  • Urinary Bladder / cytology*
  • Urinary Bladder / microbiology*
  • Urothelium / microbiology
  • rho GTP-Binding Proteins / physiology

Substances

  • Bacterial Toxins
  • CCNB1 protein, human
  • Cyclin B
  • Cyclin B1
  • Cytotoxins
  • Escherichia coli Proteins
  • Growth Inhibitors
  • cytotoxic necrotizing factor type 1
  • rho GTP-Binding Proteins