Impaired postnatal development in C/EBPbeta-deficient mice

J Reprod Dev. 2006 Oct;52(5):645-9. doi: 10.1262/jrd.17093. Epub 2006 Jul 13.

Abstract

Tail DNA genotyping of fetal and neonatal mice from C/EBPbeta heterozygous parents was performed to determine whether the decreased number of surviving C/EBPbeta mutants was caused by prenatal or postnatal death. Eighty-four 3-week-old mice born of heterozygous parents had significantly lower numbers of C/EBPbeta-deficient offspring than the expected Mendelian ratio (29.8%+/+, 65.5%+/-, 4.76%-/-, P<0.05). The genotypes of 72 fetal mice from intercrossed heterozygotes showed approximately the expected 1:2:1 Mendelian ratio (18.1% +/+, 52.8% +/-, 29.2% -/-, P>0.05). No difference in the proportions by sex could be detected in these perinates. This data indicates that C/EBPbeta-deficient mice have unknown lethal problems between the embryonic stage and weaning.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • CCAAT-Enhancer-Binding Protein-beta / deficiency*
  • CCAAT-Enhancer-Binding Protein-beta / genetics
  • Female
  • Genes, Lethal / physiology*
  • Genotype
  • Male
  • Mice
  • Mice, Mutant Strains / genetics
  • Mice, Mutant Strains / growth & development*

Substances

  • CCAAT-Enhancer-Binding Protein-beta