Advantage of serum type-I arginase and ornithine carbamoyltransferase in the evaluation of acute and chronic liver damage induced by thioacetamide in rats

Clin Chim Acta. 2007 Jan;375(1-2):63-8. doi: 10.1016/j.cca.2006.06.018. Epub 2006 Jun 21.

Abstract

Background: We evaluated the usefulness of serum type-I arginase (ARG) and ornithine carbamoyltransferase (OCT) in thioacetamide (TAA)-induced acute and chronic liver injury in rats.

Methods: In an acute injury model, we measured the time-courses of serum concentrations of ARG and OCT using ELISA, together with AST and ALT using biochemical enzymatic assays after a single administration of TAA (200 mg/kg, i.p.). In the chronic model, TAA was repeatedly administered (20 mg/kg/day, p.o.) for 16 weeks and serum concentrations of the enzymes were evaluated.

Results: In the acute model, the concentrations of the enzymes were increased in a similar manner, peaking 24 h after the administration, and ARG showed the earliest and greatest increase among the enzymes tested. In the chronic model, the serum concentration of OCT was significantly increased only 1 week after oral treatment, while concentrations of the other enzymes were increased at 8 to 12 weeks. In the histological analysis, TAA treatment damaged hepatocytes in both the acute and chronic model.

Conclusions: These results clearly show the usefulness of ARG and OCT for the evaluation of acute and chronic liver injury, respectively.

MeSH terms

  • Alanine Transaminase / blood
  • Animals
  • Arginase / blood*
  • Aspartate Aminotransferases / blood
  • Biomarkers / blood
  • Chemical and Drug Induced Liver Injury
  • Disease Models, Animal
  • Liver / drug effects
  • Liver / enzymology
  • Liver / pathology
  • Liver Diseases / blood*
  • Liver Diseases / enzymology
  • Male
  • Ornithine Carbamoyltransferase / blood*
  • Rats
  • Rats, Wistar
  • Thioacetamide

Substances

  • Biomarkers
  • Thioacetamide
  • Ornithine Carbamoyltransferase
  • Aspartate Aminotransferases
  • Alanine Transaminase
  • Arginase