Prophylactic aspirin therapy does not increase faecal calprotectin concentrations

Eur J Gastroenterol Hepatol. 2006 Sep;18(9):965-7. doi: 10.1097/01.meg.0000230080.59135.e1.

Abstract

Objective: Non-steroidal anti-inflammatory drugs (NSAID) can induce enteropathy. Aspirin ingestion is associated with a lower small-intestinal inflammation than other NSAID. Faecal calprotectin concentrations have recently been proposed as a simple non-invasive test to identify NSAID enteropathy. The aim of our pilot study was to evaluate calprotectin concentrations in patients on treatment with low-dose aspirin.

Methods: Twenty-two patients on prophylactic treatment with aspirin were recruited. Twenty-five healthy volunteers were enrolled as a control group. Faecal calprotectin concentrations were determined by enzyme-linked immunosorbent assay. Statistical analysis was performed by t-test for unpaired data.

Results: The mean faecal calprotectin concentration in patients (57.95+/-44.28 microg/g) did not show significant differences compared with controls (45.76+/-26.45 microg/g; P=0.251).

Conclusions: We found that low-dose aspirin does not induce an increase in faecal calprotectin increase.

MeSH terms

  • Anti-Inflammatory Agents, Non-Steroidal / adverse effects*
  • Aspirin / adverse effects*
  • Biomarkers / analysis
  • Carotid Artery Diseases / drug therapy
  • Case-Control Studies
  • Enteritis / chemically induced*
  • Feces / chemistry*
  • Female
  • Humans
  • Leukocyte L1 Antigen Complex / analysis*
  • Male
  • Middle Aged
  • Pilot Projects

Substances

  • Anti-Inflammatory Agents, Non-Steroidal
  • Biomarkers
  • Leukocyte L1 Antigen Complex
  • Aspirin