Progesterone-modulated phosphatidylserine externalization in apoptosis and activation of Jurkat cells

Am J Reprod Immunol. 2006 Oct;56(4):249-57. doi: 10.1111/j.1600-0897.2006.00422.x.

Abstract

Problem: During pregnancy the elevated levels of progesterone (Pg) have immunomodulating effects. It is important to follow-up Pg effects on basic biological processes at cell level as apoptosis and activation which was the aim of this study.

Methods of study: Jurkat cells cultured in the presence or absence of Pg were used as a model system. Apoptosis was induced by H(2)O(2) and activation by phorbol myriastate acetate. The induced changes in the phosphatidylserine (PS) externalization and cell surface CD69 expression were followed by fluorescence-activated cell sorter and immunofluorescence.

Results: After the induction of apoptosis PS externalizes in 52.3% of Jurkat cells. Cells cultured with Pg show tendency to a decrease of PS positive cells (42%). The opposite effect is observed in activated cells--PS externalization increase from 33.8% of control cells to 40.1% of Pg-treated cells.

Conclusions: These findings would suggest that by increasing activation and decreasing apoptosis Pg could regulate local immune system during pregnancy.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / physiology*
  • Biological Transport, Active / physiology
  • Humans
  • Jurkat Cells
  • Lymphocyte Activation / immunology
  • Lymphocyte Activation / physiology*
  • Phosphatidylserines / metabolism*
  • Progesterone / physiology*

Substances

  • Phosphatidylserines
  • Progesterone