Expression of gene rrg is associated with reversion of NIH 3T3 transformed by LTR-c-H-ras

Science. 1990 Aug 17;249(4970):796-8. doi: 10.1126/science.1697103.

Abstract

A partial complementary DNA was isolated for a gene (rrg) that is normally expressed in mouse NIH 3T3 cells, but is down-regulated after cellular transformation by long terminal repeat (LTR)-activated c-H-ras (LTR-c-H-ras). This gene was reexpressed in a nontumorigenic persistent revertant cell line created by prolonged treatment of the transformed cells with mouse interferon alpha/beta. Persistent revertants stably transfected with rrg complementary DNA antisense expression vectors appeared transformed, had decreased amounts of rrg messenger RNA, and were tumorigenic in nude mice. Stable transfection with sense constructs did not alter the normal morphology, message level, or nontumorigenicity of the persistent revertant cell line.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cell Line, Transformed
  • Cloning, Molecular
  • DNA / genetics
  • Gene Expression*
  • Genes, ras*
  • Humans
  • Interferon Type I / pharmacology
  • Mice
  • Mice, Nude
  • Neoplasms, Experimental / etiology
  • Nucleic Acid Hybridization
  • Proto-Oncogene Proteins / genetics*
  • Proto-Oncogene Proteins p21(ras)
  • RNA / analysis
  • RNA / genetics
  • RNA, Antisense
  • RNA, Messenger / genetics
  • Rats
  • Transfection

Substances

  • Interferon Type I
  • Proto-Oncogene Proteins
  • RNA, Antisense
  • RNA, Messenger
  • RNA
  • DNA
  • HRAS protein, human
  • Proto-Oncogene Proteins p21(ras)

Associated data

  • GENBANK/M35796