Multifactorial control of insulin receptor gene expression in human cell lines

Biochem Biophys Res Commun. 1990 Aug 16;170(3):1184-90. doi: 10.1016/0006-291x(90)90518-r.

Abstract

Taking advantage of a computer program to align DNA sequences we scanned the 5'-flanking region of the insulin receptor gene to localize putative regulatory elements. Using this approach as a start point, we investigated the effect of several agents on insulin receptor gene expression in human cells. We found that (i) forskolin reduces insulin receptor mRNA levels; (ii) the effects of either forskolin or dexamethasone on insulin receptor mRNA levels are near completely abolished in glucose-starved cells; (iii) 2-mercaptoethanol and heat shock, besides previously described stress conditions (glycosylatyon inhibitors), reduce insulin receptor mRNA levels.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • Blotting, Northern
  • Cell Line
  • Colforsin / pharmacology
  • Cyclic AMP / physiology
  • DNA / analysis*
  • Dexamethasone / pharmacology
  • Gene Expression Regulation* / drug effects
  • Hot Temperature
  • Humans
  • Mercaptoethanol / pharmacology
  • Molecular Sequence Data
  • Promoter Regions, Genetic
  • RNA / analysis
  • Receptor, Insulin / drug effects
  • Receptor, Insulin / genetics*
  • Sequence Homology, Nucleic Acid

Substances

  • Colforsin
  • Mercaptoethanol
  • RNA
  • Dexamethasone
  • DNA
  • Cyclic AMP
  • Receptor, Insulin