A traditional Japanese medicine mitigates TNBS-induced colitis in rats

Scand J Gastroenterol. 2006 Oct;41(10):1183-9. doi: 10.1080/00365520600575704.

Abstract

Objective: Differences in dietary habits may be one of the reasons that the incidence of inflammatory bowel disease has remained lower in Japan than in Western countries. We investigated whether a traditional Japanese medicine (Strong Wakamto), based on Aspergillus oryzae koji, would exert any effect on experimental colitis in rats.

Material and methods: Colitis was induced using an enema of trinitrobenzene sulfonic acid (TNBS) and ethanol. Strong Wakamto was administered for 28 days before induction of colitis and for 7 days thereafter, and the effect of this medicine was evaluated.

Results: Treatment with 5% Strong Wakamto improved loss of body-weight, increased colon weight and significantly decreased the histological damage score for colon mucosa. Decreases in faecal Lactobacillus sp., superoxide dismutase activity and zinc concentrations, and the increased IL-1beta expression in colonic tissue after TNBS enema were improved when Strong Wakamto was given. The present in vitro studies indicate that administration of Strong Wakamto prevents lipopolysaccharide-induced TNF-alpha production in human macrophages.

Conclusions: Oral administration of Strong Wakamto mitigates experimental inflammatory bowel disease induced by TNBS enema in rats. The beneficial effects seem attributable to a combination of balancing microflora, immunomodulatory effects on gut macrophages, and enhancement of anti-superoxide activity in colonic tissues.

MeSH terms

  • Animals
  • Aspergillus oryzae*
  • Body Weight
  • Colitis / chemically induced
  • Colitis / drug therapy*
  • Colon / metabolism
  • Enema*
  • Feces / microbiology
  • In Vitro Techniques
  • Interleukin-1beta / analysis
  • Intestinal Mucosa / pathology
  • Japan
  • Macrophages / metabolism
  • Male
  • Medicine, East Asian Traditional*
  • Organ Size
  • Rats
  • Rats, Sprague-Dawley
  • Superoxide Dismutase / analysis
  • Trinitrobenzenesulfonic Acid*
  • Tumor Necrosis Factor-alpha / biosynthesis
  • Zinc / analysis

Substances

  • Interleukin-1beta
  • Tumor Necrosis Factor-alpha
  • Trinitrobenzenesulfonic Acid
  • Superoxide Dismutase
  • Zinc