The effects of chronic tea intake on platelet activation and inflammation: a double-blind placebo controlled trial

Atherosclerosis. 2007 Aug;193(2):277-82. doi: 10.1016/j.atherosclerosis.2006.08.054. Epub 2006 Sep 29.

Abstract

Background: Tea drinking appears to protect against the development of coronary heart disease (CHD), but the mediating pathways are uncertain. We studied the effects of 6 weeks of black tea or placebo on platelet activation, C-reactive protein (CRP), total antioxidant status, and soluble (s) P-Selectin in a randomized double-blind trial.

Methods: Healthy non-smoking men aged 18-55 years were randomized to black tea (N=37) or placebo (N=38) following a 4-week washout period during which they drank no tea, coffee or caffeinated beverages, but consumed caffeinated placebo tea. Bloods were drawn after 6 weeks of treatment. Platelet activation was assessed by measuring leukocyte-platelet aggregates using whole blood flow cytometry.

Results: Following treatment, the tea group had fewer monocyte-platelet aggregates (means 5.84 versus 6.60%, P=0.027), neutrophil-platelet aggregates (P=0.017), total leukocyte-platelet aggregates (P=0.027), and lower plasma C-reactive protein (means 0.76 versus 0.97 mg/L, P=0.05) than the placebo group. There were no differences in total antioxidant status or soluble P-Selectin.

Conclusions: Chronic tea consumption reduces platelet activation and plasma C-reactive protein in healthy men. Effects cannot be attributed to observer bias or lifestyle confounders. These effects of tea may contribute to sustained cardiovascular health.

Publication types

  • Randomized Controlled Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Antioxidants / analysis
  • Beverages*
  • C-Reactive Protein / analysis
  • Double-Blind Method
  • Humans
  • Inflammation
  • Male
  • Middle Aged
  • Oxidative Stress / drug effects*
  • P-Selectin / blood
  • Platelet Activation / drug effects*
  • Tea*

Substances

  • Antioxidants
  • P-Selectin
  • Tea
  • C-Reactive Protein