POMGnT1 mutations in congenital muscular dystrophy: genotype-phenotype correlation and expanded clinical spectrum

Arch Neurol. 2006 Oct;63(10):1491-5. doi: 10.1001/archneur.63.10.1491.

Abstract

Background: Muscle-eye-brain disease is a congenital muscular dystrophy with eye and brain involvement due to POMGnT1 mutations.

Objective: To describe the clinical and molecular features of 3 Italian patients with POMGnT1 mutations.

Design: Case reports.

Patients: One patient had muscle and brain abnormalities without eye involvement. Two patients had a classic muscle-eye-brain disease phenotype with different levels of clinical severity.

Results: Brain magnetic resonance imaging showed cortical malformation and posterior fossa involvement. Immunofluorescence for glycosylated alpha-dystroglycan performed on muscle biopsy specimens demonstrated an absent signal in 1 patient and reduced staining in 2 patients. Molecular analysis identified 5 mutations, 2 of which are novel.

Conclusion: This article adds to what is known about the genotype-phenotype correlation and expands our awareness of the clinical spectrum associated with POMGnT1 mutations.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Brain / abnormalities
  • Brain / metabolism
  • Brain / physiopathology
  • Child
  • Conserved Sequence / genetics
  • DNA Mutational Analysis
  • Dystroglycans / metabolism
  • Female
  • Genetic Predisposition to Disease / genetics*
  • Genotype
  • Glycation End Products, Advanced / genetics
  • Glycation End Products, Advanced / metabolism
  • Humans
  • Magnetic Resonance Imaging
  • Male
  • Muscle, Skeletal / metabolism
  • Muscle, Skeletal / pathology
  • Muscle, Skeletal / physiopathology
  • Muscular Dystrophies / congenital*
  • Muscular Dystrophies / genetics*
  • Muscular Dystrophies / physiopathology
  • Mutation / genetics*
  • N-Acetylglucosaminyltransferases / genetics*
  • Phenotype
  • Retina / metabolism
  • Retina / physiopathology
  • Sequence Homology, Nucleic Acid
  • Syndrome

Substances

  • Glycation End Products, Advanced
  • Dystroglycans
  • N-Acetylglucosaminyltransferases
  • protein O-mannose beta-1,2-N-acetylglucosaminyltransferase