Possible involvement of GTP-binding proteins in the deactivation of an inwardly rectifying K+ current in enterocytes isolated from guinea-pig small intestine

Pflugers Arch. 1990 Oct;417(2):240-2. doi: 10.1007/BF00370706.

Abstract

The possible regulation of guinea pig enterocyte ion channels by GTP-binding proteins has been investigated by using the whole-cell recording mode of the patch-clamp technique and non-hydrolysable analogues of GTP. The main K+ currents in these cells are mediated by inwardly-rectifying K+ channels. Intracellular dialysis with hydrolysis-resistant GTP analogues leads to the deactivation of the inward K+ currents. Non-hydrolysable analogues of ATP or GDP were without effect. The effect occurs after a lag phase of 2 to 7 min, suggesting a multistep mechanism. Cl currents were not affected by any of the nucleotide analogues. It is suggested that inwardly-rectifying K+ currents are deactivated by a G-protein-dependent process.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Chlorides / metabolism
  • GTP-Binding Proteins / metabolism*
  • Guanosine 5'-O-(3-Thiotriphosphate) / pharmacology
  • Guinea Pigs
  • In Vitro Techniques
  • Intestinal Mucosa / cytology
  • Intestinal Mucosa / metabolism*
  • Intestine, Small / cytology
  • Intestine, Small / metabolism
  • Ion Channels / metabolism
  • Potassium Channels / metabolism*

Substances

  • Chlorides
  • Ion Channels
  • Potassium Channels
  • Guanosine 5'-O-(3-Thiotriphosphate)
  • GTP-Binding Proteins