Abstract
A fraction of primitive "lymphoid" precursors retain plasticity for myeloid differentiation. In this issue of Immunity, Laiosa et al. describe that Notch-Delta signals can protect thymic precursors from reprogramming into the myeloid lineage, antagonizing the enforced myeloid transcription factors such as PU.1 and C/EBPalpha.
MeSH terms
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Animals
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CCAAT-Enhancer-Binding Protein-alpha / immunology
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CCAAT-Enhancer-Binding Protein-alpha / metabolism
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Cell Differentiation / immunology*
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Cell Lineage / immunology*
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Hematopoietic Stem Cells / immunology
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Humans
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Proto-Oncogene Proteins / immunology
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Proto-Oncogene Proteins / metabolism
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Receptors, Notch / immunology
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Receptors, Notch / metabolism
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Receptors, Opioid, delta / immunology
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Receptors, Opioid, delta / metabolism
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T-Lymphocytes / cytology*
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T-Lymphocytes / immunology*
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Thymus Gland / cytology*
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Thymus Gland / immunology
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Trans-Activators / immunology
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Trans-Activators / metabolism
Substances
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CCAAT-Enhancer-Binding Protein-alpha
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Proto-Oncogene Proteins
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Receptors, Notch
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Receptors, Opioid, delta
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Trans-Activators
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proto-oncogene protein Spi-1