PAR-1 activation has different effects on the angiogenic activity of endothelial progenitor cells derived from human adult and cord blood

J Thromb Haemost. 2006 Dec;4(12):2729-31. doi: 10.1111/j.1538-7836.2006.02208.x.
No abstract available

Publication types

  • Comparative Study
  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult Stem Cells / cytology
  • Adult Stem Cells / drug effects
  • Adult Stem Cells / metabolism*
  • Angiogenic Proteins / genetics
  • Angiogenic Proteins / metabolism
  • Cell Differentiation
  • Cell Proliferation / drug effects
  • Cell Shape
  • Cells, Cultured
  • Endothelial Cells / cytology
  • Endothelial Cells / drug effects
  • Endothelial Cells / metabolism*
  • Fetal Blood / cytology
  • Fetal Stem Cells / cytology
  • Fetal Stem Cells / drug effects
  • Fetal Stem Cells / metabolism*
  • Gene Expression Regulation / drug effects
  • Humans
  • Neovascularization, Physiologic*
  • Peptide Fragments / pharmacology
  • RNA, Messenger / metabolism
  • Receptor, PAR-1 / agonists
  • Receptor, PAR-1 / metabolism*
  • Receptors, Cell Surface / genetics
  • Receptors, Cell Surface / metabolism
  • Time Factors

Substances

  • Angiogenic Proteins
  • Peptide Fragments
  • RNA, Messenger
  • Receptor, PAR-1
  • Receptors, Cell Surface
  • thrombin receptor peptide (42-47)