Imatinib mesylate induces hypophosphatemia in patients with chronic myeloid leukemia in late chronic phase, and this effect is associated with response

Am J Hematol. 2007 May;82(5):394-5. doi: 10.1002/ajh.20778.

Abstract

Imatinib is the drug of first choice for most patients with chronic phase chronic myeloid leukemia (CML). Although it is generally well tolerated, a number of hematological and nonhematological side-effects have been described. We report here that imatinib induces hypophosphatemia in a high proportion of our series of CML patients previously treated with interferon alpha, and that this previously unreported side effect is associated with response.

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Benzamides
  • Calcium / blood
  • Cohort Studies
  • Female
  • Humans
  • Hypophosphatemia / chemically induced*
  • Imatinib Mesylate
  • Interferon-alpha / therapeutic use
  • Kidney Tubules / drug effects*
  • Kidney Tubules / metabolism
  • Leukemia, Myeloid, Chronic-Phase / blood
  • Leukemia, Myeloid, Chronic-Phase / drug therapy*
  • Male
  • Middle Aged
  • Parathyroid Hormone / blood
  • Phosphates / metabolism
  • Piperazines / adverse effects
  • Piperazines / pharmacology*
  • Piperazines / therapeutic use
  • Pyrimidines / adverse effects
  • Pyrimidines / pharmacology*
  • Pyrimidines / therapeutic use
  • Retrospective Studies
  • Treatment Outcome

Substances

  • Benzamides
  • Interferon-alpha
  • Parathyroid Hormone
  • Phosphates
  • Piperazines
  • Pyrimidines
  • Imatinib Mesylate
  • Calcium