Hyperbaric oxygenation attenuates renal ischemia-reperfusion injury in rats

Transplant Proc. 2006 Dec;38(10):3337-40. doi: 10.1016/j.transproceed.2006.10.184.

Abstract

Background: Oxygen-derived free radicals play an important role in ischemia-reperfusion injury (IR). Hyperbaric oxygenation (HO) decreases free radical production. The aim of this study was to determine the effect of HO treatment on renal ischemia-reperfusion injury in rats.

Methods: Rats were divided into four groups. All groups underwent right nephrectomy. Group I served as the control group; group II had left renal ischemia-reperfusion; group III was pretreated with HO; and group IV, ischemia-reperfusion and HO pretreatment. Tissue malondialdehyde (MDA) and glutathione (GSH) levels were measured, and histopathologic damage scored.

Results: HO pretreatment significantly decreased tissue MDA levels and histopathologic scores among rats with IR. There was an increased GSH in HO-pretreated rats with IR; however, the difference was not significant.

Conclusion: HO prior to ischemia displayed a beneficial effect on renal IR by reducing oxygen radical peroxidation of lipid membranes.

MeSH terms

  • Animals
  • Blood Urea Nitrogen
  • Creatinine / blood
  • Disease Models, Animal
  • Hyperbaric Oxygenation*
  • Male
  • Necrosis
  • Rats
  • Rats, Sprague-Dawley
  • Renal Circulation
  • Reperfusion Injury / blood
  • Reperfusion Injury / pathology
  • Reperfusion Injury / physiopathology
  • Reperfusion Injury / prevention & control*

Substances

  • Creatinine