Regulation of memory antibody levels: the role of persisting antigen versus plasma cell life span

J Immunol. 2007 Jan 1;178(1):67-76. doi: 10.4049/jimmunol.178.1.67.

Abstract

Protective Ab levels can be maintained for years upon infection or vaccination. In this study, we studied the duration of Ab responses as a function of the life span of plasma cells and tested the role of persisting Ag in maintaining B cell memory. Our analysis of B cell responses induced in mice immunized with virus-like particles demonstrates the following: 1) Ab titers are long-lived, but decline continuously with a t(1/2) of approximately 80 days, which corresponds to the life span of plasma cells; 2) the germinal center (GC) reaction, which lasts for up to 100 days, is dependent on Ag associated with follicular dendritic cells; and 3) early GCs produce massive numbers of plasma and memory B cell precursors, whereas the late Ag-dependent GCs are dispensable for the maintenance of Ab levels and B cell memory.

MeSH terms

  • Allolevivirus / immunology
  • Animals
  • Antibodies / blood*
  • Antigens / blood
  • Antigens / immunology*
  • Cell Proliferation
  • Dendritic Cells, Follicular / drug effects
  • Dendritic Cells, Follicular / immunology
  • Germinal Center / cytology
  • Germinal Center / immunology
  • Immunization
  • Immunoglobulins / pharmacology
  • Immunologic Memory*
  • Immunosuppression Therapy
  • Kinetics
  • Lymphotoxin beta Receptor / immunology
  • Mice
  • Mice, Inbred C57BL
  • Plasma Cells / cytology
  • Plasma Cells / immunology*
  • Virion / immunology

Substances

  • Antibodies
  • Antigens
  • Immunoglobulins
  • Lymphotoxin beta Receptor