Functional arterial and venous fate is determined by graded VEGF signaling and notch status during embryonic stem cell differentiation

Arterioscler Thromb Vasc Biol. 2007 Mar;27(3):487-93. doi: 10.1161/01.ATV.0000255990.91805.6d. Epub 2006 Dec 21.

Abstract

Objective: The aim of this work was to develop a mouse embryonic stem (ES) cell system addressing the early specification of the developing vasculature into functional arteries and veins.

Methods and results: ES cells were differentiated 4 days on collagen-type IV coated dishes to obtain Flk1+ endothelial precursors. Sub-culture of these precursors for additional 4 days robustly generated, in a VEGF dose-dependent manner, mature endothelial cells. Arterial marker genes were specifically expressed in cultures differentiated with high VEGF concentration whereas the venous marker gene COUP-TFII was upregulated in endothelial cells induced through low and intermediate VEGF concentrations. This VEGF-dependent arterialization could be blocked by inhibition of Notch resulting in an arterial to venous fate switch. Functional and morphological studies, ie, measurement of sprout length, pericyte recruitment, and interleukin-I-induced leukocyte adhesion, further confirmed their arterial and venous identity.

Conclusions: We conclude that endothelial cells with distinct molecular, morphological, and functional characteristics of arteries and veins can be derived through in vitro differentiation of ES cells in a VEGF dose-dependent and Notch-regulated manner.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Arteries / embryology*
  • Cell Differentiation
  • Cells, Cultured
  • Embryonic Stem Cells
  • Endothelium, Vascular / embryology
  • Gene Expression Regulation, Developmental
  • Immunohistochemistry
  • Mice
  • Neovascularization, Physiologic / physiology
  • Probability
  • RNA / analysis
  • Receptors, Notch / metabolism*
  • Receptors, Notch / physiology
  • Reverse Transcriptase Polymerase Chain Reaction
  • Sensitivity and Specificity
  • Signal Transduction / physiology*
  • Vascular Endothelial Growth Factor A / metabolism*
  • Veins / embryology*

Substances

  • Receptors, Notch
  • Vascular Endothelial Growth Factor A
  • RNA