Improved glucose tolerance in acyl CoA:diacylglycerol acyltransferase 1-null mice is dependent on diet

Lipids Health Dis. 2007 Jan 19:6:2. doi: 10.1186/1476-511X-6-2.

Abstract

Background: Mice that lack acyl CoA:diacylglycerol acyltransferase (Dgat1-/- mice) are reported to have a reduced body fat content and improved glucose tolerance and insulin sensitivity. Studies so far have focussed on male null mice fed a high fat diet and there are few data on heterozygotes. We compared male and female Dgat1-/-, Dgat1+/- and Dgat1+/+ C57Bl/6 mice fed on either standard chow or a high fat diet.

Results: Body fat content was lower in the Dgat1-/- than the Dgat1+/+ mice in both experiments; lean body mass was higher in male Dgat1-/- than Dgat1+/+ mice fed on the high fat diet. Energy intake and expenditure were higher in male Dgat1-/- than Dgat1+/+ mice; these differences were less marked or absent in females. The body fat content of female Dgat1+/- mice was intermediate between that of Dgat1-/- and Dgat1+/+ mice, whereas male Dgat1+/- mice were similar to or fatter than Dgat1+/+ mice. Glucose tolerance was improved and plasma insulin reduced in Dgat1-/- mice fed on the high fat diet, but not on the chow diet. Both male and female Dgat1+/- mice had similar glucose tolerance to Dgat1+/+ mice.

Conclusion: These results suggest that although ablation of DGAT1 improves glucose tolerance by preventing obesity in mice fed on a high fat diet, it does not improve glucose tolerance in mice fed on a low fat diet.

MeSH terms

  • Adipose Tissue / anatomy & histology
  • Animal Feed
  • Animals
  • Blood Glucose / metabolism*
  • Body Weight
  • Diacylglycerol O-Acyltransferase / deficiency*
  • Diet*
  • Energy Intake
  • Female
  • Genotype
  • Male
  • Mice
  • Mice, Knockout

Substances

  • Blood Glucose
  • Dgat1 protein, mouse
  • Diacylglycerol O-Acyltransferase