Cardiovascular protection with sildenafil following chronic inhibition of nitric oxide synthase

Br J Pharmacol. 2007 Mar;150(5):538-40. doi: 10.1038/sj.bjp.0707132. Epub 2007 Jan 22.

Abstract

During the past 18 years, sildenafil has evolved from a potential anti-angina drug to an on-demand treatment for erectile dysfunction and more recently to a new orally active treatment for pulmonary hypertension. Recent studies suggest that the drug has powerful cardioprotective effect against ischemia/reperfusion injury, doxorubicin-induced cardiomyopathy and anti-hypertensive effect induced by chronic inhibition of nitric oxide synthase in animals. Based on several recent basic and clinical studies, it is clear that sildenafil and other clinically approved type-5 phosphodiesterase-5 inhibitors including vardenafil and tadalafil will eventually be developed for several cardiovascular indications including essential hypertension, endothelial dysfunction, ischemia/reperfusion injury, myocardial infarction, ventricular remodeling and heart failure.

Publication types

  • Comment
  • Research Support, N.I.H., Extramural

MeSH terms

  • 3',5'-Cyclic-GMP Phosphodiesterases / antagonists & inhibitors
  • Animals
  • Antihypertensive Agents / pharmacology
  • Carbolines / pharmacology
  • Cardiomyopathies / chemically induced
  • Cardiomyopathies / prevention & control
  • Cardiovascular Agents / pharmacology*
  • Cardiovascular Agents / therapeutic use
  • Cyclic Nucleotide Phosphodiesterases, Type 5
  • Disease Models, Animal
  • Doxorubicin
  • Endothelium, Vascular / drug effects
  • Enzyme Inhibitors
  • Erectile Dysfunction / drug therapy
  • Heart Failure / drug therapy
  • Humans
  • Hypertension / chemically induced
  • Hypertension / metabolism
  • Hypertension / prevention & control*
  • Hypertension, Pulmonary / drug therapy
  • Imidazoles / pharmacology
  • Male
  • Myocardial Infarction / drug therapy
  • Myocardial Reperfusion Injury / chemically induced
  • Myocardial Reperfusion Injury / metabolism
  • Myocardial Reperfusion Injury / prevention & control*
  • NG-Nitroarginine Methyl Ester
  • Nitric Oxide / metabolism*
  • Phosphodiesterase Inhibitors / pharmacology
  • Piperazines / pharmacology*
  • Piperazines / therapeutic use
  • Purines / pharmacology
  • Purines / therapeutic use
  • Sildenafil Citrate
  • Sulfones / pharmacology*
  • Sulfones / therapeutic use
  • Tadalafil
  • Triazines / pharmacology
  • Vardenafil Dihydrochloride
  • Vasodilator Agents / pharmacology
  • Ventricular Remodeling / drug effects

Substances

  • Antihypertensive Agents
  • Carbolines
  • Cardiovascular Agents
  • Enzyme Inhibitors
  • Imidazoles
  • Phosphodiesterase Inhibitors
  • Piperazines
  • Purines
  • Sulfones
  • Triazines
  • Vasodilator Agents
  • Nitric Oxide
  • Vardenafil Dihydrochloride
  • Tadalafil
  • Doxorubicin
  • Sildenafil Citrate
  • 3',5'-Cyclic-GMP Phosphodiesterases
  • Cyclic Nucleotide Phosphodiesterases, Type 5
  • PDE5A protein, human
  • NG-Nitroarginine Methyl Ester