Heme oxygenase-1 and interleukin-11 are overexpressed in stress-induced premature senescence of human WI-38 fibroblasts induced by tert-butylhydroperoxide and ethanol

Biogerontology. 2007 Aug;8(4):409-22. doi: 10.1007/s10522-007-9084-8. Epub 2007 Feb 13.

Abstract

Acute repeated exposures to subcytotoxic concentrations of tert-butylhydroperoxide and ethanol trigger premature senescence of human diploid fibroblasts. In the present work we found an increased mRNA and protein level of interleukin-11 and heme oxygenase-1 in premature senescence of WI-38 human diploid foetal lung fibroblasts induced by both tert-butylhydroperoxide and ethanol. We tested whether interleukin-11 and heme oxygenase-1 could protect against tert-butylhydroperoxide- or ethanol-induced premature senescence when stable overexpression was established using a retroviral vector-based transduction. No protective effect was found against the decrease of the proliferative potential, the increase of the proportion of senescence-associated ss-galactosidase positive cells and the increase of the mRNA levels of six senescence-associated genes.

MeSH terms

  • Cell Line
  • Cell Proliferation / drug effects
  • Cellular Senescence / drug effects*
  • Cellular Senescence / genetics
  • Enzyme Induction
  • Ethanol / pharmacology*
  • Fibroblasts / drug effects*
  • Fibroblasts / enzymology
  • Fibroblasts / metabolism
  • Heme Oxygenase-1 / biosynthesis*
  • Heme Oxygenase-1 / genetics
  • Humans
  • Interleukin-11 / biosynthesis*
  • Interleukin-11 / genetics
  • Lung / drug effects*
  • Lung / embryology
  • Lung / metabolism
  • Oxidative Stress / drug effects*
  • Oxidative Stress / genetics
  • RNA, Messenger / biosynthesis
  • Transduction, Genetic
  • Up-Regulation
  • beta-Galactosidase / metabolism
  • tert-Butylhydroperoxide / pharmacology*

Substances

  • IL11 protein, human
  • Interleukin-11
  • RNA, Messenger
  • Ethanol
  • tert-Butylhydroperoxide
  • HMOX1 protein, human
  • Heme Oxygenase-1
  • beta-Galactosidase