Expression of p16, CD95, CD95L and Helix pomatia agglutinin in relapsing and nonrelapsing very thin melanoma

Br J Dermatol. 2007 Mar;156(3):440-7. doi: 10.1111/j.1365-2133.2006.07581.x.

Abstract

Background: The incidence of malignant melanoma is increasing worldwide and patients are being diagnosed earlier with thinner primary lesions. Most patients with very thin melanoma (Breslow thickness < 0.76 mm) are cured by surgery but 2-18% relapse locally or with distant metastases.

Objectives: The objective of this study was to establish potential new prognostic markers in very thin melanoma.

Methods: We identified a group of subjects with relapsing very thin primary cutaneous melanoma and a matched control group who had not relapsed. We investigated the expression of p16, Helix pomatia agglutinin (HPA), CD95 and CD95 ligand (CD95L) by immunohistochemistry on paraffin-embedded tissue sections from the subject group, their subsequent metastases and the control group.

Results: Reduced p16 expression was significantly associated with relapse in very thin melanoma (P = 0.0129). Loss of p16 expression was also found in 76% of metastases. There was no significant association between HPA, CD95 or CD95L expression and subsequent relapse.

Conclusions: This work is the first to show a significant loss of p16 in relapsing very thin melanoma.

Publication types

  • Multicenter Study

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Biomarkers, Tumor / metabolism*
  • Cyclin-Dependent Kinase Inhibitor p16 / metabolism
  • Fas Ligand Protein / metabolism
  • Female
  • Humans
  • Lectins / metabolism
  • Male
  • Melanoma / metabolism*
  • Melanoma / pathology
  • Melanoma / secondary
  • Middle Aged
  • Neoplasm Proteins / metabolism
  • Severity of Illness Index
  • Skin Neoplasms / metabolism*
  • Skin Neoplasms / pathology
  • fas Receptor / metabolism

Substances

  • Biomarkers, Tumor
  • Cyclin-Dependent Kinase Inhibitor p16
  • Fas Ligand Protein
  • Helix lectin
  • Lectins
  • Neoplasm Proteins
  • fas Receptor