Cutting Edge: TFII-I controls B cell proliferation via regulating NF-kappaB

J Immunol. 2007 Mar 1;178(5):2631-5. doi: 10.4049/jimmunol.178.5.2631.

Abstract

The multifunctional transcription factor TFII-I physically and functionally interacts with Bruton's tyrosine kinase in murine B cells. However, the downstream functions of TFII-I in B cells are unknown. Toward achieving this goal, we established stable posttranscriptional silencing of TFII-I in WEHI-231 immature murine B cells, which undergoes growth arrest and apoptosis either upon anti-IgM or TGF-beta signaling. In this study, we show that TFII-I promotes growth arrest of cells in a signal-dependent manner. Unlike control cells, B cells exhibiting loss of TFII-I function fail to undergo arrest upon signaling due to up-regulation of c-Myc expression and concomitant down-regulation of both p21 and p27. Loss of TFII-I is also associated with simultaneous increase in nuclear c-rel and decrease in p50 homodimer binding. Thus, besides controlling c-myc transcription, TFII-I controls B cell proliferation by regulating both nuclear translocation of c-rel and DNA-binding activity of p50 NF-kappaB.

MeSH terms

  • Active Transport, Cell Nucleus / genetics
  • Active Transport, Cell Nucleus / immunology
  • Animals
  • Apoptosis / genetics
  • Apoptosis / immunology
  • B-Lymphocytes / cytology
  • B-Lymphocytes / immunology*
  • B-Lymphocytes / metabolism
  • Cell Line
  • Cell Nucleus / genetics
  • Cell Nucleus / immunology
  • Cell Nucleus / metabolism
  • Cell Proliferation*
  • Down-Regulation / genetics
  • Down-Regulation / immunology
  • Immunoglobulin M / immunology
  • Mice
  • NF-kappa B p50 Subunit / genetics
  • NF-kappa B p50 Subunit / immunology*
  • NF-kappa B p50 Subunit / metabolism
  • Proto-Oncogene Proteins c-rel / genetics
  • Proto-Oncogene Proteins c-rel / immunology*
  • Proto-Oncogene Proteins c-rel / metabolism
  • Signal Transduction / genetics
  • Signal Transduction / immunology*
  • Transcription Factors, TFII / genetics
  • Transcription Factors, TFII / immunology*
  • Transcription Factors, TFII / metabolism
  • Transforming Growth Factor beta / immunology
  • Up-Regulation / genetics
  • Up-Regulation / immunology

Substances

  • Immunoglobulin M
  • NF-kappa B p50 Subunit
  • Proto-Oncogene Proteins c-rel
  • Transcription Factors, TFII
  • Transforming Growth Factor beta