The Intestinal Wnt/TCF Signature

Gastroenterology. 2007 Feb;132(2):628-32. doi: 10.1053/j.gastro.2006.08.039. Epub 2006 Aug 18.

Abstract

Background & aims: In colorectal cancer, activating mutations in the Wnt pathway transform epithelial cells through the inappropriate expression of a TCF4 target gene program, which is physiologically expressed in intestinal crypts.

Methods: We have now performed an exhaustive array-based analysis of this target gene program in colorectal cancer cell lines carrying an inducible block of the Wnt cascade. Independently, differential gene-expression profiles of human adenomas and adenocarcinomas vs normal colonic epithelium were obtained.

Results: Expression analyses of approximately 80 genes common between these data sets were performed in a murine adenoma model. The combined data sets describe a core target gene program, the intestinal Wnt/TCF signature gene set, which is responsible for the transformation of human intestinal epithelial cells.

Conclusions: The genes were invariably expressed in adenomas, yet could be subdivided into 3 modules, based on expression in distinct crypt compartments. A module of 17 genes was specifically expressed at the position of the crypt stem cell.

MeSH terms

  • Adenocarcinoma / genetics
  • Adenocarcinoma / metabolism
  • Adenoma / genetics
  • Adenoma / metabolism
  • Animals
  • Cell Line, Tumor
  • Cell Transformation, Neoplastic / genetics
  • Cell Transformation, Neoplastic / metabolism*
  • Cell Transformation, Neoplastic / pathology
  • Colorectal Neoplasms / genetics
  • Colorectal Neoplasms / metabolism
  • Gene Expression Profiling
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Intestinal Mucosa / metabolism*
  • Intestinal Mucosa / pathology
  • Mice
  • Oligonucleotide Array Sequence Analysis
  • Paneth Cells / metabolism
  • RNA, Messenger / metabolism
  • Signal Transduction* / genetics
  • T Cell Transcription Factor 1 / metabolism
  • TCF Transcription Factors / genetics
  • TCF Transcription Factors / metabolism*
  • Time Factors
  • Transcription Factor 7-Like 2 Protein
  • Transfection
  • Wnt Proteins / genetics
  • Wnt Proteins / metabolism*
  • beta Catenin / metabolism

Substances

  • RNA, Messenger
  • T Cell Transcription Factor 1
  • TCF Transcription Factors
  • TCF7L2 protein, human
  • Tcf7l2 protein, mouse
  • Transcription Factor 7-Like 2 Protein
  • Wnt Proteins
  • beta Catenin