Mucinous cystadenocarcinoma of the pancreas developing during hormone replacement therapy

Dig Dis Sci. 2007 May;52(5):1326-8. doi: 10.1007/s10620-006-9482-9. Epub 2007 Mar 20.

Abstract

Hormone replacement therapy (HRT) containing estrogens is generally used to relieve climacteric symptoms and to prevent osteoporosis and coronary heart disease [1], however, there has been increasing evidence of the HRT as the risk of hormone-dependent neoplasms including breast cancer [2], uterine endometrial cancer [3], ovarian cancer [4], and even lung cancer [5]. Noteworthy is mucinous cyst neoplasms (MCNs) of the pancreas, characterized by mucin-producing columnar epithelium supported by "ovarian-like" mesenchymal stroma, occur mostly in females expressing estrogen receptors [6, 7]. Although several reports regarding the closed relationship between MCNs and pregnancy [8, 9] might imply potential sex hormone-dependency of the MCNs [10], no correlation has been reported. This is the first case report of malignant MCN developing during continuous HRT after hysterectomy.

Publication types

  • Case Reports

MeSH terms

  • Cystadenocarcinoma, Mucinous / chemically induced*
  • Cystadenocarcinoma, Mucinous / chemistry
  • Cystadenocarcinoma, Mucinous / pathology
  • Cystadenocarcinoma, Mucinous / surgery
  • Estrogen Replacement Therapy / adverse effects*
  • Female
  • Humans
  • Hysterectomy
  • Inhibins / analysis
  • Middle Aged
  • Neoplasms, Hormone-Dependent / chemically induced*
  • Neoplasms, Hormone-Dependent / chemistry
  • Neoplasms, Hormone-Dependent / pathology
  • Neoplasms, Hormone-Dependent / surgery
  • Pancreatectomy
  • Pancreatic Neoplasms / chemically induced*
  • Pancreatic Neoplasms / chemistry
  • Pancreatic Neoplasms / pathology
  • Pancreatic Neoplasms / surgery
  • Receptors, Estrogen / analysis
  • Receptors, Progesterone / analysis
  • Splenectomy
  • Stromal Cells / chemistry
  • Stromal Cells / drug effects*
  • Stromal Cells / pathology
  • Treatment Outcome
  • Ultrasonography / methods

Substances

  • Receptors, Estrogen
  • Receptors, Progesterone
  • Inhibins