Resistin-like molecule-beta is an allergen-induced cytokine with inflammatory and remodeling activity in the murine lung

Am J Physiol Lung Cell Mol Physiol. 2007 Aug;293(2):L305-13. doi: 10.1152/ajplung.00147.2007. Epub 2007 Jun 1.

Abstract

Resistin-like molecule (RELM)-beta is a cysteine-rich cytokine implicated in insulin resistance and asthmatic responses, but its function remains an enigma. We now report that RELM-beta has a role in promoting airway inflammation and lung remodeling in the mouse lung. RELM-beta is strongly induced by diverse allergens and T helper type 2 (Th2) cytokines by an IL-13- and STAT6-dependent mechanism. To understand the in vivo role of RELM-beta, we delivered recombinant murine RELM-beta intratracheally to naïve mice. RELM-beta induced dose-dependent leukocyte accumulation (most prominently involving macrophages) and goblet cell hyperplasia. The most prominent effect induced by RELM-beta was increased perivascular and peribronchial collagen deposition. Mice genetically deficient in RELM-beta had reduced accumulation of collagen and goblet cell hyperplasia in an experimental model of allergic airway inflammation. In vitro experiments demonstrated that RELM-beta had fibroblast motogenic activity. These results identify RELM-beta as a Th2-associated cytokine with potent inflammatory and remodeling activity.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Allergens / immunology*
  • Allergens / metabolism
  • Animals
  • Asthma / immunology
  • Asthma / metabolism
  • Bronchoalveolar Lavage Fluid / cytology
  • Bronchoalveolar Lavage Fluid / immunology
  • Cell Movement
  • Collagen / metabolism
  • Female
  • Fibroblasts / immunology
  • Fibroblasts / metabolism
  • Fibroblasts / pathology
  • Goblet Cells / immunology
  • Hormones, Ectopic / genetics
  • Hormones, Ectopic / immunology*
  • Hormones, Ectopic / pharmacology
  • Intercellular Signaling Peptides and Proteins
  • Interleukin-13 / metabolism
  • Interleukin-4 / metabolism
  • Lung / immunology*
  • Lung / metabolism
  • Lung / pathology
  • Male
  • Mice
  • Mice, Inbred BALB C
  • Mice, Mutant Strains
  • NIH 3T3 Cells
  • Pneumonia / immunology*
  • Pneumonia / metabolism
  • Pneumonia / pathology
  • Respiratory Mucosa / immunology
  • Respiratory Mucosa / metabolism
  • Respiratory Mucosa / pathology
  • STAT6 Transcription Factor / metabolism
  • Th2 Cells / drug effects
  • Th2 Cells / immunology
  • Th2 Cells / metabolism

Substances

  • Allergens
  • Hormones, Ectopic
  • Intercellular Signaling Peptides and Proteins
  • Interleukin-13
  • Retnlb protein, mouse
  • STAT6 Transcription Factor
  • Stat6 protein, mouse
  • Interleukin-4
  • Collagen