Limbic encephalitis as presentation of a SAP deficiency

Neurology. 2007 Jul 10;69(2):218-9. doi: 10.1212/01.wnl.0000265597.56202.6c.
No abstract available

Publication types

  • Case Reports

MeSH terms

  • Adolescent
  • CD8-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / pathology
  • Cerebral Arteries / pathology
  • Cerebral Arteries / physiopathology
  • Chemotaxis, Leukocyte / genetics
  • Chemotaxis, Leukocyte / immunology
  • Disease Progression
  • Epilepsy, Temporal Lobe / genetics
  • Epilepsy, Temporal Lobe / immunology
  • Epilepsy, Temporal Lobe / physiopathology
  • Fatal Outcome
  • Humans
  • Immunosuppressive Agents / therapeutic use
  • Intracellular Signaling Peptides and Proteins / deficiency*
  • Intracellular Signaling Peptides and Proteins / genetics*
  • Limbic Encephalitis / genetics*
  • Limbic Encephalitis / immunology*
  • Limbic Encephalitis / physiopathology
  • Limbic System / pathology*
  • Limbic System / physiopathology
  • Lymphoma, B-Cell / genetics
  • Lymphoma, B-Cell / immunology
  • Lymphoproliferative Disorders / complications*
  • Lymphoproliferative Disorders / genetics
  • Lymphoproliferative Disorders / immunology
  • Male
  • Signaling Lymphocytic Activation Molecule Associated Protein
  • Temporal Lobe / pathology
  • Temporal Lobe / physiopathology
  • Treatment Failure
  • Vasculitis, Central Nervous System / genetics
  • Vasculitis, Central Nervous System / immunology
  • Vasculitis, Central Nervous System / physiopathology

Substances

  • Immunosuppressive Agents
  • Intracellular Signaling Peptides and Proteins
  • SH2D1A protein, human
  • Signaling Lymphocytic Activation Molecule Associated Protein