Polymorphisms in TLR2 are associated with increased viral shedding and lesional rate in patients with genital herpes simplex virus Type 2 infection

J Infect Dis. 2007 Aug 15;196(4):505-9. doi: 10.1086/519693. Epub 2007 Jun 29.

Abstract

Clinical and virologic manifestations of genital herpes simplex virus type 2 (HSV-2) infection vary widely. We examined frequencies of single-nucleotide polymorphisms (SNPs) in Toll-like receptors (TLRs) 2, 3, 4, and 9 in a prospective cohort of 128 HSV-2-infected persons whose viral shedding and lesion frequency was measured by daily sampling from genital secretions. Two TLR2 haplotypes (2 and 4) were associated with increased lesional (P=.008 and P=.03) and shedding (P=.02 and P=.001) rates. An SNP in haplotype 2 (-15607A/G) was also associated with shedding (P=.01) and lesional (P=.008) rates. Polymorphisms in TLR2 may be in part responsible for differences in the severity of HSV-2 infection.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Cohort Studies
  • DNA, Viral / genetics
  • Disease Progression
  • Female
  • Genetic Predisposition to Disease
  • Haplotypes
  • Herpes Genitalis / genetics*
  • Herpes Genitalis / pathology
  • Herpes Genitalis / virology*
  • Herpesvirus 2, Human / genetics
  • Herpesvirus 2, Human / isolation & purification*
  • Herpesvirus 2, Human / physiology
  • Humans
  • Male
  • Polymerase Chain Reaction
  • Polymorphism, Genetic
  • Prospective Studies
  • Toll-Like Receptor 2 / genetics*

Substances

  • DNA, Viral
  • TLR2 protein, human
  • Toll-Like Receptor 2