Signal-transducing adaptor protein-2 regulates integrin-mediated T cell adhesion through protein degradation of focal adhesion kinase

J Immunol. 2007 Aug 15;179(4):2397-407. doi: 10.4049/jimmunol.179.4.2397.

Abstract

Signal-transducing adaptor protein-2 (STAP-2) is a recently identified adaptor protein that contains pleckstrin homology- and Src homology 2-like domains as well as a YXXQ motif in its C-terminal region. Our previous studies demonstrated that STAP-2 binds to STAT3 and STAT5, and regulates their signaling pathways. In the present study, we find that STAP-2-deficient splenocytes or T cells exhibit enhanced cell adhesion to fibronectin after PMA treatment, and that STAP-2-deficient T cells contain the increased protein contents of focal adhesion kinase (FAK). Furthermore, overexpression of STAP-2 induces a dramatic decrease in the protein contents of FAK and integrin-mediated T cell adhesion to fibronectin in Jurkat T cells via the degradation of FAK. Regarding the mechanism for this effect, we found that STAP-2 associates with FAK and enhances its degradation, proteasome inhibitors block FAK degradation, and STAP-2 recruits an endogenous E3 ubiquitin ligase, Cbl, to FAK. These results reveal a novel regulation mechanism for integrin-mediated signaling in T cells via STAP-2, which directly interacts with and degrades FAK.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptor Proteins, Signal Transducing / deficiency
  • Adaptor Proteins, Signal Transducing / immunology*
  • Adaptor Proteins, Signal Transducing / metabolism
  • Amino Acid Motifs / genetics
  • Amino Acid Motifs / immunology
  • Animals
  • Cell Adhesion / genetics
  • Cell Adhesion / immunology
  • Fibronectins / immunology
  • Fibronectins / metabolism
  • Focal Adhesion Kinase 1 / genetics
  • Focal Adhesion Kinase 1 / immunology*
  • Focal Adhesion Kinase 1 / metabolism
  • Humans
  • Integrins / genetics
  • Integrins / immunology
  • Integrins / metabolism
  • Jurkat Cells
  • Mice
  • Mice, Knockout
  • Phosphoproteins / deficiency
  • Phosphoproteins / immunology*
  • Phosphoproteins / metabolism
  • Proteasome Endopeptidase Complex / genetics
  • Proteasome Endopeptidase Complex / immunology*
  • Proteasome Endopeptidase Complex / metabolism
  • Proto-Oncogene Proteins c-cbl / genetics
  • Proto-Oncogene Proteins c-cbl / immunology
  • Proto-Oncogene Proteins c-cbl / metabolism
  • STAT2 Transcription Factor / genetics
  • STAT2 Transcription Factor / immunology
  • STAT2 Transcription Factor / metabolism
  • STAT3 Transcription Factor / genetics
  • STAT3 Transcription Factor / immunology
  • STAT3 Transcription Factor / metabolism
  • Signal Transduction / genetics
  • Signal Transduction / immunology*
  • T-Lymphocytes / immunology*
  • T-Lymphocytes / metabolism
  • src Homology Domains / genetics
  • src Homology Domains / immunology

Substances

  • Adaptor Proteins, Signal Transducing
  • Fibronectins
  • Integrins
  • Phosphoproteins
  • STAP2 protein, human
  • STAP2 protein, mouse
  • STAT2 Transcription Factor
  • STAT2 protein, human
  • STAT3 Transcription Factor
  • STAT3 protein, human
  • Stat2 protein, mouse
  • Stat3 protein, mouse
  • Proto-Oncogene Proteins c-cbl
  • Focal Adhesion Kinase 1
  • PTK2 protein, human
  • Ptk2 protein, mouse
  • Proteasome Endopeptidase Complex
  • CBL protein, human