Multiple forms of nuclear p53 formed in human Raji and MEC1 cells treated with fludarabine

Leukemia. 2008 Mar;22(3):657-60. doi: 10.1038/sj.leu.2404929. Epub 2007 Aug 30.
No abstract available

Publication types

  • Comparative Study
  • Letter

MeSH terms

  • Antimetabolites, Antineoplastic / pharmacology*
  • Apoptosis / drug effects
  • Burkitt Lymphoma / pathology
  • Cell Line, Tumor / drug effects
  • Cell Line, Tumor / metabolism
  • DNA Damage
  • Drug Resistance, Neoplasm / genetics
  • Genes, p53
  • Humans
  • Isoelectric Point
  • Leukemia, Lymphocytic, Chronic, B-Cell / genetics
  • Leukemia, Lymphocytic, Chronic, B-Cell / pathology
  • Molecular Weight
  • Mutation
  • Neoplasm Proteins / chemistry
  • Neoplasm Proteins / drug effects*
  • Neoplasm Proteins / genetics
  • Peptide Fragments / chemistry
  • Peptide Fragments / metabolism
  • Peptide Hydrolases / metabolism
  • Phosphorylation / drug effects
  • Protein Processing, Post-Translational / drug effects
  • Protein Structure, Tertiary
  • Tumor Suppressor Protein p53 / chemistry
  • Tumor Suppressor Protein p53 / drug effects*
  • Vidarabine / analogs & derivatives*
  • Vidarabine / pharmacology

Substances

  • Antimetabolites, Antineoplastic
  • Neoplasm Proteins
  • Peptide Fragments
  • Tumor Suppressor Protein p53
  • Peptide Hydrolases
  • Vidarabine
  • fludarabine