[Expression of erythropoietin receptor in esophageal carcinoma and its correlation to microvessel density]

Ai Zheng. 2008 Jan;27(1):52-7.
[Article in Chinese]

Abstract

Background & objective: Erythropoietin receptor (EPO-R) is expressed in many kinds of tumors. The EPO/EPO-R signaling is involved in tumor cell proliferation, angiogenesis, and invasion. This study was to detect the expression of EPO-R in esophageal carcinoma, and explore its correlation to angiogenesis.

Methods: The expression of EPO-R protein in 110 specimens of primary esophageal carcinoma was assessed by immunohistochemistry. The microvessels were immunohistochemically labeled with factor VIII-related antigen (FVIII-R Ag) to assess microvessel density (MVD).

Results: EPO-R was detected in all tumor tissues. It was expressed in cytoplasm and/or on cell membrane. Up-regulated EPO-R expression was correlated to poor differentiation (P<0.001) and lymph node metastasis (P=0.006). Increased MVD was correlated to poor differentiation (P<0.001), lymph node metastasis (P<0.001), advanced stage (III/IVA/IVB) (P=0.022), and smoking (P=0.029). The expression of EPO-R was positively correlated to MVD (r=0.618, P<0.01).

Conclusions: EPO-R expressed generally in esophageal carcinoma. The level of EPO-R positively correlates to angiogenesis and progression of esophageal carcinoma. EPO might be an endogenous stimulant of angiogenesis during the progression of esophageal carcinoma.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Age Factors
  • Carcinoma, Squamous Cell / blood supply
  • Carcinoma, Squamous Cell / metabolism*
  • Carcinoma, Squamous Cell / pathology
  • Esophageal Neoplasms / blood supply
  • Esophageal Neoplasms / metabolism*
  • Esophageal Neoplasms / pathology
  • Female
  • Humans
  • Immunohistochemistry
  • Lymphatic Metastasis
  • Male
  • Microvessels / pathology*
  • Middle Aged
  • Neoplasm Staging
  • Neovascularization, Pathologic / metabolism*
  • Neovascularization, Pathologic / pathology
  • Receptors, Erythropoietin / metabolism*
  • Sex Factors
  • Smoking
  • von Willebrand Factor / metabolism

Substances

  • Receptors, Erythropoietin
  • von Willebrand Factor