Glutamine decreases intestinal nuclear factor kappa B activity and pro-inflammatory cytokine expression after traumatic brain injury in rats

Inflamm Res. 2008 Feb;57(2):57-64. doi: 10.1007/s00011-007-7101-7.

Abstract

Objective: To investigate whether glutamine supplementation modulates intestinal nuclear factor kappa B (NF-kappaB) activity and pro-inflammatory cytokine expression after traumatic brain injury (TBI) in rats.

Materials and methods: Right parietal cortical contusion in male rats was made by the weight-dropping method. After trauma, the rats were randomly given chow alone or glutamine mixed chow for 5 d. Gut samples were extracted at 5 d postinjury. We measured NF-kappaB binding activity by electrophoretic mobility shift assay; NF-kappaB subunits p50 and p65 expression by immunohistochemistry; the concentrations of interleukin-1beta, tumor necrosis factor-alpha and interleukin-6 by enzyme-linked immunosorbent assay; intestinal mucosal morphological changes by histopathological study and electron microscopy; and apoptosis by terminal deoxynucleotidyl transferase-mediated dUTP nick end labeling staining.

Results: Administration of glutamine following TBI could decrease NF-kappaB binding activity, NF-kappaB p65 protein expression and concentrations of pro-inflammatory cytokines in the gut. TBI-induced damage of gut structure was ameliorated after glutamine supplementation.

Conclusion: The results of the present study suggest that the therapeutic benefit of post-TBI glutamine supplementation might be due to its inhibitory effects on intestinal NF-kappaB activation and pro-inflammatory cytokine expression.

MeSH terms

  • Animals
  • Apoptosis
  • Brain Injuries / metabolism*
  • Cell Nucleus / metabolism
  • Cytokines / metabolism*
  • Gene Expression Regulation*
  • Glutamine / pharmacology*
  • Interleukin-1beta / metabolism
  • Interleukin-6 / metabolism
  • Intestinal Mucosa / metabolism*
  • Intestines / drug effects
  • Male
  • NF-kappa B / metabolism*
  • Rats
  • Rats, Wistar
  • Transcription Factor RelA / metabolism
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Cytokines
  • Interleukin-1beta
  • Interleukin-6
  • NF-kappa B
  • Rela protein, rat
  • Transcription Factor RelA
  • Tumor Necrosis Factor-alpha
  • Glutamine