Natural killer cells, glutathione, cytokines, and innate immunity against Mycobacterium tuberculosis

J Interferon Cytokine Res. 2008 Mar;28(3):153-65. doi: 10.1089/jir.2007.0095.

Abstract

It is becoming increasingly apparent that natural killer (NK) cells play a crucial role in innate defense mechanisms against Mycobacterium tuberculosis infection. Furthermore, NK cell functions are dependent on adequate levels of glutathione. In this study, we examined whether the NK cell-mediated growth control of intracellular M. tuberculosis is dependent on adequate levels of glutathione. We investigated the effects of glutathione both alone and in combination with interleukin-2 (IL-2) or IL-12 or both in modulating NK cell functions, such as cytolytic activity, activating receptor expression, induction of apoptosis, and cytokine synthesis. Our results strongly indicate that glutathione in combination with IL-2+IL-12 augments NK cell functions, leading to control M. tuberculosis infection.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis
  • Cell Line
  • Cytokines / immunology
  • Cytokines / metabolism*
  • Glutathione / metabolism*
  • Glutathione / pharmacology
  • Humans
  • Immunity, Innate
  • Interleukin-12 / immunology
  • Interleukin-12 / metabolism*
  • Interleukin-2 / immunology
  • Interleukin-2 / metabolism*
  • Killer Cells, Natural / immunology*
  • Monocytes / immunology
  • Monocytes / metabolism*
  • Mycobacterium tuberculosis / drug effects
  • Mycobacterium tuberculosis / immunology*
  • Protein Array Analysis

Substances

  • Cytokines
  • Interleukin-2
  • Interleukin-12
  • Glutathione