[A novel mutation in the SEDL gene leading to X-linked spondyloepiphyseal dysplasia tarda in a large Chinese pedigree]

Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 2008 Apr;25(2):150-3.
[Article in Chinese]

Abstract

Objective: To identify the genetic defect in a four-generation pedigree with X-linked recessive spondyloepiphyseal dysplasia tarda (SEDT) from Southwest China.

Methods: Linkage analysis with one panel of fluorescently labeled microsatellite markers on chromosome X and mutation screening of SEDL gene by direct sequencing were performed.

Results: Linkage between SEDT and Xp22.2-Xp23.1 was established with maximum LOD score of 3.82 (theta = 0) between DXS987 and DXS8051. Upon sequence analysis, a point mutation within exon 4 of the SEDL gene (c.239A to G) was found which resulted in substitution of histidine with arginine at codon 80 (His80Arg).

Conclusion: A novel missense mutation (H80R) was identified for SEDL gene in the large Chinese SEDT pedigree.

MeSH terms

  • Adolescent
  • Adult
  • Asian People / genetics
  • DNA Mutational Analysis
  • Female
  • Genetic Diseases, X-Linked / genetics*
  • Genetic Diseases, X-Linked / pathology
  • Genetic Linkage / genetics
  • Humans
  • Male
  • Membrane Transport Proteins / genetics*
  • Mutation
  • Mutation, Missense / genetics
  • Osteochondrodysplasias / genetics*
  • Osteochondrodysplasias / pathology
  • Pedigree
  • Transcription Factors / genetics*

Substances

  • Membrane Transport Proteins
  • TRAPPC2 protein, human
  • Transcription Factors