Increased anxiety-like behavior in neuropsin (kallikrein-related peptidase 8) gene-deficient mice

Behav Neurosci. 2008 Jun;122(3):498-504. doi: 10.1037/0735-7044.122.3.498.

Abstract

Neuropsin (kallikrein-related peptidase 8) is concentrated in the hippocampus, amygdala, olfactory bulb, and prefrontal cortex. Earlier studies showed that protease deficiency causes a significant impairment of early-phase long-term potentiation in the Schaffer collateral pathway and hippocampus-dependent memory in the Y maze and Morris water maze (Z. Chen et al., 1995; A. Hirata et al., 2001; H. Tamura et al., 2006). In addition to neuropsin's participation in the hippocampal memory, amygdalar and cortical localization of the gene suggests extrahippocampal behavioral function, and the authors therefore examined neuropsin-deficient mice, including tests of sensory motor reflex, open field, light-dark transition, Rota-Rod, elevated plus-maze, hot plate, startle response-prepulse inhibition, Porsolt forced swim, Barnes maze, eight-arm radial maze, and contextual and cued fear conditioning tests. Here, the authors found increased anxiety in neuropsin-deficient mice, suggesting the involvement of this protease in emotional responses.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acoustic Stimulation / methods
  • Analysis of Variance
  • Animals
  • Anxiety / genetics*
  • Anxiety / physiopathology*
  • Avoidance Learning / physiology
  • Behavior, Animal / physiology*
  • Conditioning, Psychological / physiology
  • Cues
  • Exploratory Behavior / physiology
  • Fear / physiology
  • Kallikreins / deficiency*
  • Male
  • Maze Learning
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Motor Activity / genetics
  • Neural Inhibition / genetics
  • Pain Measurement
  • Psychomotor Performance / physiology
  • Reflex, Startle / genetics

Substances

  • Kallikreins