Caveolin-3 deficiency decreases the gene expression level of osteopontin in mdx mouse skeletal muscle

Acta Myol. 2006 Oct;25(2):53-61.

Abstract

Caveolin-3 is a muscle-specific membrane protein that serves as a scaffold of various molecules. As previously reported, caveolin-3 deficiency causes muscle degeneration in mice. In the present study, gene expression profiles, analyzed in the skeletal muscles of caveolin-3 deficient mice using the DNA microarray technique, showed that the gene of osteopontin, a versatile regulator of inflammation and tissue repair, was significantly down-regulated. This is in contrast to mdx mice showing a markedly up-regulated osteopontin gene in their skeletal muscles. Recently, osteopontin has been reported to be important in the pathogenesis of muscular dystrophy. We examined whether up-regulated osteopontin gene expression in mdx muscles is altered by the deficiency of caveolin-3. To this end, we developed caveolin-3 and dystrophin double-deficient mice and used them for the analysis. Levels of osteopontin mRNA and protein in the double-deficient mice clearly decreased compared with those in mdx mice.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Caveolin 3 / deficiency*
  • Female
  • Male
  • Mice
  • Mice, Inbred mdx
  • Muscle, Skeletal / metabolism*
  • Muscle, Skeletal / pathology
  • Oligonucleotide Array Sequence Analysis
  • Osteopontin / genetics
  • Osteopontin / metabolism*
  • RNA, Messenger / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction

Substances

  • Cav3 protein, mouse
  • Caveolin 3
  • RNA, Messenger
  • Spp1 protein, mouse
  • Osteopontin