Smurf2 is a TRAF2 binding protein that triggers TNF-R2 ubiquitination and TNF-R2-induced JNK activation

Biochem Biophys Res Commun. 2008 Oct 3;374(4):752-7. doi: 10.1016/j.bbrc.2008.07.103. Epub 2008 Jul 29.

Abstract

TRAF2 plays a central role in TNF-induced signalling to NF-kappaB and JNK/p38 MAPK. To better understand the molecular mechanisms that mediate this dual function of TRAF2, we performed a yeast two-hybrid screening for TRAF2 interacting proteins using the Sos recruitment system. This resulted in the identification of the E3 ubiquitin ligase Smurf2 as a TRAF2 binding protein. TRAF2 overexpression was shown to trigger Smurf2 ubiquitination and the formation of a TNF-R2/Smurf2 complex. Smurf2 on its turn promoted TNF-R2 ubiquitination and the relocalization of TNF-R2 as well as TRAF2 to a detergent-insoluble cell fraction. This was associated with enhanced TNF-R2-induced JNK activation, whereas TNF-R2-induced NF-kappaB activation remained unaffected. These results suggest an important role for Smurf2 binding to TRAF2 in determining specific signalling outputs of TNF-R2.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Line
  • Enzyme Activation
  • Humans
  • JNK Mitogen-Activated Protein Kinases / metabolism*
  • Receptors, Tumor Necrosis Factor, Type II / metabolism*
  • TNF Receptor-Associated Factor 2 / metabolism*
  • Two-Hybrid System Techniques
  • Ubiquitin-Protein Ligases / metabolism*
  • Ubiquitination*

Substances

  • Receptors, Tumor Necrosis Factor, Type II
  • TNF Receptor-Associated Factor 2
  • SMURF2 protein, human
  • Ubiquitin-Protein Ligases
  • JNK Mitogen-Activated Protein Kinases