Effects of transforming growth factor-beta 1 on growth of aortic smooth muscle cells. Influences of interaction with growth factors, cell state, cell phenotype, and cell cycle

Atherosclerosis. 1991 Jun;88(2-3):227-34. doi: 10.1016/0021-9150(91)90085-h.

Abstract

Transforming growth factor (TGF)-beta 1 may have different effects on cell proliferation depending on many conditions. This paper clarifies the effects of various conditions on the effect of TGF-beta 1 on proliferation of cultured rabbit aortic smooth muscle cells (SMC) and also the time of its action during the cell cycle. TGF-beta 1 at 10-10,000 pg/ml inhibited DNA synthesis of SMC in the G0 stage derived from normal media or atheromatous intima stimulated by either platelet-derived growth factor (PDGF), fibroblast growth factor, SMC-derived growth factor, or fetal bovine serum (FBS). TGF-beta 1 also inhibited the growth of SMC in the growing state stimulated by either PDGF or FBS. TGF-beta 1 was effective only when added to the culture within 2 h after stimulation of the G0 state SMC with PDGF. It also inhibited increase in transcription of the c-myc protooncogene on stimulation of SMC with PDGF. These data suggest that TGF-beta 1 inhibited proliferation of SMC irrespective of the cell phenotype, growth conditions, and growth factors present and that it exerted this inhibitory effect during the time of the G0/G1 transition.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Aorta, Thoracic
  • Cell Cycle / drug effects
  • Cell Division / drug effects
  • Cells, Cultured
  • DNA / biosynthesis
  • Fetal Blood
  • Fibroblast Growth Factors / pharmacology
  • Growth Substances / pharmacology*
  • Immunoblotting
  • Muscle, Smooth, Vascular / cytology*
  • Muscle, Smooth, Vascular / drug effects
  • Muscle, Smooth, Vascular / metabolism
  • Phenotype
  • Platelet-Derived Growth Factor / pharmacology
  • Proto-Oncogene Proteins c-myc / genetics
  • Rabbits
  • Suppression, Genetic / drug effects
  • Transforming Growth Factor beta / pharmacology*

Substances

  • Growth Substances
  • Platelet-Derived Growth Factor
  • Proto-Oncogene Proteins c-myc
  • Transforming Growth Factor beta
  • Fibroblast Growth Factors
  • DNA