The effect of montelukast on atherogenesis in apoE/LDLR-double knockout mice

J Physiol Pharmacol. 2008 Sep;59(3):633-9.

Abstract

We have shown that inhibitors of five lipoxygenase activating protein (FLAP)--MK-886 and BAYx1005 inhibit atherosclerosis in apolipoprotein E/LDL receptor-double knockout mice. We, therefore, investigated whether cysteinyl leukotrienes receptor inhibitor--montelukast, given at a dose of 0.125 microg per 100 mg of body weight per day during 16 weeks, could also attenuate atherogenesis. In apoE/LDLR-DKO mouse model montelukast significantly decreased atherogenesis, measured both by "en face" method (25.5+/-2.% vs. 17.23 +/- 1.8%) and "cross-section" method (455,494 +/- 26,477 microm(2) vs. 299,201 +/- 20,373 microm(2)). The results were, however, less pronounced, comparing to FLAP inhibitors. This is the first report showing the effect of montelukast on atherogenesis in gene-targeted mice.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetates / therapeutic use*
  • Animals
  • Aorta / pathology
  • Apolipoproteins E / genetics*
  • Atherosclerosis / blood
  • Atherosclerosis / pathology
  • Atherosclerosis / prevention & control*
  • Cholesterol / blood
  • Cyclopropanes
  • Female
  • Leukotriene Antagonists / therapeutic use*
  • Lipids / blood
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Myocardium / pathology
  • Quinolines / therapeutic use*
  • Receptors, LDL / genetics*
  • Sulfides
  • Triglycerides / blood

Substances

  • Acetates
  • Apolipoproteins E
  • Cyclopropanes
  • Leukotriene Antagonists
  • Lipids
  • Quinolines
  • Receptors, LDL
  • Sulfides
  • Triglycerides
  • Cholesterol
  • montelukast