Abstract
In this issue of Molecular Cell, Ditzel et al. (2008) show that DIAP1 polyubiquitinates DRONC, DCP-1, and drICE, leading to their nondegradative inactivation. Surprisingly, activation of DIAP1 requires caspase-mediated cleavage, revealing an elegant feedback mechanism by which caspases regulate their own fate.
MeSH terms
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Animals
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Apoptosis / genetics
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Apoptosis / physiology*
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Caspases / genetics
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Caspases / metabolism*
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Cell Survival
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Drosophila / cytology
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Drosophila / genetics
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Drosophila / metabolism
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Feedback, Physiological
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Inhibitor of Apoptosis Proteins / genetics
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Inhibitor of Apoptosis Proteins / metabolism*
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Models, Biological
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Ubiquitination
Substances
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Inhibitor of Apoptosis Proteins
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Caspases