Inhibition of COX-2 and activation of peroxisome proliferator-activated receptor gamma synergistically inhibits proliferation and induces apoptosis of human pancreatic carcinoma cells

Cancer Lett. 2009 Mar 18;275(2):247-55. doi: 10.1016/j.canlet.2008.10.023. Epub 2008 Dec 3.

Abstract

Although inhibition of cyclooxygenase-2 (COX-2) or activation of peroxisome proliferators-activated receptor gamma (PPAR-gamma) leads to growth inhibition in malignancies, the synergistic anti-tumor effects of combination of COX-2 inhibitor (NS-398) and PPAR-gamma agonist (rosiglitazone) on the human pancreatic cancer cells remains unknown. Here, we evaluated the effects of NS-398 and/or rosiglitazone on the cell proliferation and apoptosis in a pancreatic cancer cell line, SW1990. NS-398 and rosiglitazone decreased cell proliferation in a dose- and time-dependent manner. Proliferating cell nuclear antigen (PCNA) labeling index significantly decreased in the cells treated with either NS-398 or rosiglitazone. Both NS-398 and rosiglitazone alone induced apoptotic cell death of SW1990. The combination of NS-398 and rosiglitazone exerted synergistic effects on proliferation inhibition, and apoptosis induction in SW1990 cells, with down-regulation of Bcl-2 and up-regulation of Bax expression. Our results indicate that simultaneous targeting of COX-2 and PPAR-gamma inhibits pancreatic cancer development more effectively than targeting each molecule alone.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / drug effects*
  • Base Sequence
  • Cell Line, Tumor
  • Cell Proliferation / drug effects*
  • Cyclooxygenase 2 / drug effects*
  • Cyclooxygenase Inhibitors / pharmacology*
  • DNA Primers
  • Electrophoresis, Polyacrylamide Gel
  • Humans
  • Immunohistochemistry
  • Nitrobenzenes / pharmacology
  • PPAR gamma / agonists*
  • Pancreatic Neoplasms / enzymology
  • Pancreatic Neoplasms / metabolism
  • Pancreatic Neoplasms / pathology*
  • Proliferating Cell Nuclear Antigen / metabolism
  • Rosiglitazone
  • Sulfonamides / pharmacology
  • Thiazolidinediones / pharmacology

Substances

  • Cyclooxygenase Inhibitors
  • DNA Primers
  • Nitrobenzenes
  • PPAR gamma
  • Proliferating Cell Nuclear Antigen
  • Sulfonamides
  • Thiazolidinediones
  • Rosiglitazone
  • N-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide
  • Cyclooxygenase 2