[Analysis of the GABRG2 gene mutation in a Chinese family with generalized epilepsy with febrile seizures plus]

Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 2008 Dec;25(6):611-5.
[Article in Chinese]

Abstract

Objective: To identify the mutation of the GABA(A)-receptor gamma 2 subunit gene (GABRG2) in a Chinese family with generalized epilepsy with febrile seizures plus (GEFS+ ) and analyze the genotype-phenotype correlations and its inheritance.

Methods: Genomic DNA was extracted from peripheral blood lymphocytes of the proband and other available members in the GEFS+ family. The coding regions and flanking intronic regions of the GABRG2 gene were screened for mutations using polymerase chain reaction (PCR) and direct DNA sequencing.

Results: There were 7 affected members in the three-generation family, in which one with febrile seizures (FS) and six with febrile seizures plus (FS+ ). This family was consistent with the diagnostic criteria of GEFS+ . The nonsense mutation c.1287G to A (p.W390X) in the GABRG2 gene was initially identified in the proband. Seven affected members (6 FS+ and 1 FS) and one unaffected member carried the mutation. The nonsense mutation c.1287G to A/p.W390X in the GABRG2 gene was co-segregated with the GEFS+ family. The penetrance rate was about 87.5%(7/8).

Conclusion: This GEFS+ family was consistent with autosomal dominant inheritance with incomplete penetrance. GABRG2 mutation is also a disease-causing mutation in Chinese GEFS+ patients. The p.W390X mutation has not been reported previously.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Asian People / genetics*
  • Base Sequence
  • Child
  • Conserved Sequence
  • DNA Mutational Analysis
  • Epilepsy, Generalized / complications*
  • Epilepsy, Generalized / genetics*
  • Exons / genetics
  • Genotype
  • Humans
  • Male
  • Molecular Sequence Data
  • Pedigree
  • Phenotype
  • Receptors, GABA-A / chemistry
  • Receptors, GABA-A / genetics*
  • Seizures, Febrile / complications*
  • Seizures, Febrile / genetics*

Substances

  • GABRG2 protein, human
  • Receptors, GABA-A