Targeting the MHC class II pathway of antigen presentation enhances immunogenicity and safety of allergen immunotherapy

Allergy. 2009 Jan;64(1):172-8. doi: 10.1111/j.1398-9995.2008.01812.x. Epub 2008 Nov 28.

Abstract

Background: Current s.c. allergen-specific immunotherapy (SIT) leads to amelioration of IgE-mediated allergy, but it requires numerous allergen injections over several years and is frequently associated with severe side-effects. The aim of this study was to test whether modified recombinant allergens can improve therapeutic efficacy in SIT while reducing allergic side-effects.

Methods: The major cat allergen Fel d 1 was fused to a TAT-derived protein translocation domain and to a truncated invariant chain for targeting the MHC class II pathway (MAT-Fel d 1). The immunogenicity was evaluated in mice, while potential safety issues were assessed by cellular antigen stimulation test (CAST) using basophils from cat-dander-allergic patients.

Results: MAT-Fel d 1 enhanced induction of Fel d 1-specific IgG2a antibody responses as well as the secretion of IFN-gamma and IL-2 from T cells. Subcutaneous allergen-specific immunotherapy of mice using the modified Fel d 1 provided stronger protection against anaphylaxis than SIT with unmodified Fel d 1, and MAT-Fel d 1 caused less degranulation of human basophils than native Fel d 1.

Conclusion: MAT-Fel d 1 allergen enhanced protective antibody and Th1 responses in mice, while reducing human basophil degranulation. Immunotherapy using MAT-Fel d 1 allergen therefore has the potential to enhance SIT efficacy and safety, thus, shortening SIT. This should increase patient compliance and lower treatment costs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Allergens / therapeutic use
  • Animals
  • Antibody Formation / drug effects*
  • Antigen Presentation*
  • Basophils
  • Cats
  • Cell Degranulation / drug effects
  • Drug Delivery Systems
  • Drug-Related Side Effects and Adverse Reactions
  • Glycoproteins / pharmacology
  • Glycoproteins / therapeutic use
  • Histocompatibility Antigens Class II / metabolism*
  • Humans
  • Immunotherapy / methods*
  • Mice
  • Recombinant Proteins
  • Th1 Cells

Substances

  • Allergens
  • Glycoproteins
  • Histocompatibility Antigens Class II
  • Recombinant Proteins
  • Fel d 1 protein, Felis domesticus