B-cell activation during HIV-1 infection. III. Down-regulating effect of mitogens

AIDS. 1991 Jul;5(7):821-8. doi: 10.1097/00002030-199107000-00005.

Abstract

Spontaneous in vitro production of HIV-1-specific antibodies, a hallmark of infected subjects, is often down-regulated by the addition of pokeweed mitogen. We observed that a decrease in such ongoing anti-HIV-1 antibody synthesis could also be induced in cultures from most patients by addition of phytohemagglutinin and Concanavalin A, but not by Epstein-Barr virus, a selective B-cell mitogen. In most cases, this down-regulatory effect of mitogens was evident within the first 24 h of culture. The observed mitogen-associated decrease in spontaneous antibody synthesis was prevented by treating peripheral blood mononuclear cells with agents inhibiting non-major histocompatibility complex-restricted cytotoxic activity or by adding third-party cells to the cultures. In most cases, the mitogen-induced effect was also counteracted by removal of T lymphocytes or CD8+ T-cell sub-population. These findings recall a similar phenomenon observed in normal subjects following intentional immunization, and indicate that mitogen-induced down-regulation of spontaneous in vitro anti-HIV-1-antibody production most probably occurs through a lectin-dependent cytotoxic effect on activated B cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, Differentiation, T-Lymphocyte
  • B-Lymphocytes / immunology*
  • CD8 Antigens
  • Cell Separation
  • Cells, Cultured
  • Cytotoxicity Tests, Immunologic
  • Down-Regulation
  • HIV Antibodies / biosynthesis*
  • HIV Infections / immunology*
  • Humans
  • Immunoglobulins / biosynthesis
  • Kinetics
  • Leucine / analogs & derivatives
  • Leucine / pharmacology
  • Lymphocyte Activation*
  • Pokeweed Mitogens / pharmacology*
  • T-Lymphocytes / immunology

Substances

  • Antigens, Differentiation, T-Lymphocyte
  • CD8 Antigens
  • HIV Antibodies
  • Immunoglobulins
  • Pokeweed Mitogens
  • leucine methyl ester
  • Leucine