Methylation and deamination of CpGs generate p53-binding sites on a genomic scale

Trends Genet. 2009 Feb;25(2):63-6. doi: 10.1016/j.tig.2008.11.005. Epub 2008 Dec 26.

Abstract

The formation of transcription-factor-binding sites is an important evolutionary process. Here, we show that methylation and deamination of CpG dinucleotides generate in vivo p53-binding sites in numerous Alu elements and in non-repetitive DNA in a species-specific manner. In light of this, we propose that the deamination of methylated CpGs constitutes a universal mechanism for de novo generation of various transcription-factor-binding sites in Alus.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • Binding Sites
  • CpG Islands / physiology*
  • DNA Methylation*
  • Deamination
  • Genome*
  • Humans
  • Molecular Sequence Data
  • Tumor Suppressor Protein p53 / metabolism*

Substances

  • Tumor Suppressor Protein p53