Oxidative DNA damage in retinopathy of prematurity

Eur J Ophthalmol. 2009 Jan-Feb;19(1):80-5. doi: 10.1177/112067210901900112.

Abstract

Purpose: This study examines the levels of oxidative damage in patients with retinopathy of prematurity (ROP).

Methods: Fifty patients were recruited with a birthweight below 1500 g or gestational age below 32 weeks. The cases were classified into those who developed ROP (n=25) and those without ROP (n=25). The authors obtained blood and urine samples from each infant, for measuring 8-hydroxy 2-deoxyguanosine (8-OHdG) and malondialdehyde (MDA) levels, at the time of the first examination at 4-6 postnatal weeks.

Results: A significant difference was observed in leukocyte and urine 8-OHdG levels in patients with ROP compared to those without ROP (p<0.001 for both). Similarly, a significant difference was observed in plasma and urine MDA levels in patients with ROP compared to those without ROP (p<0.001 for both). In addition, significant correlations were found between levels of 8-OHdG in leukocyte DNA and plasma MDA (r=0.859, p<0.001), and between levels of urine 8-OHdG excretion and urine MDA (r=0.563, p<0.001).

Conclusions: 8-OHdG in leukocyte DNA and urine levels in premature infants can be useful as an indicator for ROP screening.

MeSH terms

  • 8-Hydroxy-2'-Deoxyguanosine
  • Biomarkers / blood
  • Biomarkers / urine
  • Birth Weight
  • Chromatography, High Pressure Liquid
  • DNA Damage*
  • Deoxyguanosine / analogs & derivatives
  • Deoxyguanosine / blood
  • Gestational Age
  • Humans
  • Infant
  • Infant, Newborn
  • Infant, Very Low Birth Weight
  • Leukocytes / metabolism
  • Lipid Peroxidation
  • Malondialdehyde / blood
  • Malondialdehyde / urine
  • Neonatal Screening / methods
  • Oxidative Stress*
  • Retinopathy of Prematurity / diagnosis*

Substances

  • Biomarkers
  • Malondialdehyde
  • 8-Hydroxy-2'-Deoxyguanosine
  • Deoxyguanosine