Abstract
A micro-array analysis using biopsies from patients with EBV-positive undifferentiated nasopharyngeal carcinoma (NPC) and from cancer-free controls revealed down-regulation of tumour suppressor genes (TSG) not previously associated with this disease; one such gene was the ataxia telangiectasia mutated (ATM) gene. Q-PCR confirmed down-regulation of ATM mRNA and ATM protein expression in tumour cells was weak or absent in almost all cases. In NPC cell lines, however, ATM was down-regulated only in the EBV-positive line, C666.1, and in none of five EBV-negative lines. In vitro infection of EBV-negative NPC cell lines with a recombinant EBV was followed by the down-regulation of ATM mRNA and protein, and only EBV-positive cells showed a defective DNA damage response following gamma-irradiation. Our data suggest that loss of ATM function could be an important step in the pathogenesis of NPC, and may have implications for the treatment of this disease.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Adenocarcinoma / genetics*
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Adenocarcinoma / virology
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Ataxia Telangiectasia Mutated Proteins
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Case-Control Studies
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Cell Cycle Proteins / analysis
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Cell Cycle Proteins / genetics*
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Cell Line, Tumor
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DNA-Binding Proteins / analysis
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DNA-Binding Proteins / genetics*
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Down-Regulation*
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Gene Expression Profiling / methods
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Gene Expression Regulation, Neoplastic*
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Gene Expression Regulation, Viral*
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Herpesvirus 4, Human
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Humans
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Immunohistochemistry
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Nasopharyngeal Neoplasms / genetics*
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Nasopharyngeal Neoplasms / virology
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Oligonucleotide Array Sequence Analysis
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Protein Serine-Threonine Kinases / analysis
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Protein Serine-Threonine Kinases / genetics*
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Reverse Transcriptase Polymerase Chain Reaction
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Tumor Suppressor Proteins / analysis
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Tumor Suppressor Proteins / genetics*
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Tumor Virus Infections / complications
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Tumor Virus Infections / genetics
Substances
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Cell Cycle Proteins
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DNA-Binding Proteins
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Tumor Suppressor Proteins
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ATM protein, human
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Ataxia Telangiectasia Mutated Proteins
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Protein Serine-Threonine Kinases