Inhibition of phorbol ester-induced cell activation in microgravity

Exp Cell Res. 1991 Nov;197(1):82-6. doi: 10.1016/0014-4827(91)90482-a.

Abstract

T lymphocytes and monocytes were exposed to microgravity and activated to produce interleukin 2 and interleukin 1, respectively. When Jurkat T cells were triggered with monoclonal antibodies directed against the CD3/T cell receptor complex in the presence of THP-1 monocytes used as accessory cells, cell-to-cell contacts took place in microgravity leading to normal production of interleukin 2 and interleukin 1, as compared to ground controls. In contrast, when cells were individually stimulated by soluble substances including a protein kinase C activating phorbol ester, the production of interleukin 1 and interleukin 2 was dramatically inhibited during microgravity exposure. This result indicates that microgravity may affect the cellular target of phorbol ester.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Calcimycin / pharmacology
  • Interleukin-1 / biosynthesis
  • Interleukin-2 / biosynthesis
  • Lymphocyte Activation*
  • Monocytes / physiology*
  • Protein Kinase C / metabolism
  • Receptors, Interleukin-2 / biosynthesis
  • T-Lymphocytes / physiology*
  • Tetradecanoylphorbol Acetate / pharmacology
  • Tumor Cells, Cultured
  • Weightlessness*

Substances

  • Interleukin-1
  • Interleukin-2
  • Receptors, Interleukin-2
  • Calcimycin
  • Protein Kinase C
  • Tetradecanoylphorbol Acetate