Construction and characterization of a novel superantigen fusion protein: bFGF/SEB

Cancer Invest. 2009 May;27(4):376-83. doi: 10.1080/07357900802487228.

Abstract

Background: As one of the bacterial superantigens, Staphylococcal enterotoxins (SEs) are potent activators of T cells, especially for those expressing T cell receptor V(beta) chains, and can induce the production of cytokines such as IFN-gamma, TNF-alpha, IL-1, IL-2, IL-6, IL-12, etc. Thus, SEs could be used in tumor-targeting therapy when cooperated with the vectors that can specifically recognize the tumor cells.

Materials and methods: The coding sequences of Staphylococcal enterotxin B (SEB) was amplified and fused with human basic fibroblast growth factor (bFGF). Recombinant protein SEB and fusion protein bFGF/SEB were expressed and purified. The biological activity was detected, including splenocytes proliferation, cytokine production, and cytotoxicity in tumor cells in vitro. In addition, the binding of bFGF/SEB with tumor cells and the tumor cell apoptosis were also tested by immunofluorescent technique.

Results: The fusion protein bFGF/SEB had similar biological activities compared with natural SEA and recombinant SEB, including tumor-inhibition ratio.

Conclusion: The recombinant bFGF/SEB-fusion protein was shown to retain the superantigenic activity of SEB, and might be a novel promising immunotherapeutic agent for the treatment of some carcinomas.

MeSH terms

  • Animals
  • Apoptosis / drug effects
  • Colorectal Neoplasms / drug therapy
  • Colorectal Neoplasms / immunology
  • Colorectal Neoplasms / pathology
  • Cytokines / metabolism
  • Cytotoxicity, Immunologic / drug effects
  • Dose-Response Relationship, Drug
  • Enterotoxins / genetics
  • Enterotoxins / immunology
  • Enterotoxins / pharmacology*
  • Female
  • Fibroblast Growth Factor 2 / genetics
  • Fibroblast Growth Factor 2 / immunology
  • Fibroblast Growth Factor 2 / pharmacology*
  • HT29 Cells
  • Humans
  • Immunoconjugates / pharmacology*
  • Immunotherapy / methods*
  • Lymphocyte Activation / drug effects
  • Mice
  • Mice, Inbred BALB C
  • Recombinant Fusion Proteins / pharmacology
  • Spleen / drug effects
  • Spleen / immunology
  • Superantigens / genetics
  • Superantigens / immunology
  • Superantigens / pharmacology*
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / immunology
  • Time Factors

Substances

  • Cytokines
  • Enterotoxins
  • Immunoconjugates
  • Recombinant Fusion Proteins
  • Superantigens
  • Fibroblast Growth Factor 2
  • enterotoxin B, staphylococcal